Dipentene C2CAS:68956-56-9

  • Product Name:Dipentene
  • CAS:68956-56-9
  • Molecular formula:C10H16
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Dipentene C2CAS:68956-56-9

Dipentene C2CAS:68956-56-9 Basic information

Product Name: Dipentene
Synonyms: dipentene fluka spec;Dipentene, pure, fraction of terpene hydrocarbons;DIPENTENE, FRACTION OF TERPENE HYDROCARBONS;Dipentene, fraction of terpene hydrocarbons, pure;DIPENTENE-C;LIME OIL TERPENES;GLIDSOL(R) 140;GLIDSOL(R) 175
CAS: 68956-56-9
MF: C10H16
MW: 136.24
EINECS: 273-309-3
Mol File: 68956-56-9.mol

Dipentene C2CAS:68956-56-9 Chemical Properties

density: 0.850
Boiling point: 173-190°C
form: Liquid
storage temp.: Flammables area
refractive index: n20/D 1.469
Fp: 42°C
CAS DataBase Reference: 68956-56-9
EPA Substance Registry System: Hydrocarbons, terpene processing by-products (68956-56-9)

Appearance   

Colorless to light yellow clear liquid.  

Color, APHA

70 Max

Odor

Lemon, pine

Specific Gravity, 20/4

0.840 - 0.870

Refractive Index @ 20℃

1.4600 - 1.4900

Distillation Range, % in 170 - 190℃

90 Min

Analysis

Area Percent  

Column Type

SE-54/BP-5

Column Size

50m×0.32mm×0.25um

Injector

250    

Detector

FID,  250   

Solvent

N/A

Oven Program

120 (8 min) to 220(2 min) at 5/min


Industrial Uses• Agriculture
Studies have shown that (+)-Dipentene has anti-insect[2], antibacterial[3].In addition, the study found that there was a correlation between the changes in the content of terpenes in pine trees and the damage of Pinus tabulaeformis.Limonene reduced the attraction of T. piniperda to attractant-baited traps and trap logs[4].Encapsulating blueberries with monolayer liposomes containing D-limonene can prevent fruit rot and spoilage caused by Botrytis cinerea and Penicillium, E. coli, Listeria, etc[1].
•Pharmaceutical industry
Studies have shown that (+)-Dipentene has anti-inflammatory, pain-reducing, cancer prevention and treatment effects[5]. (+)-Dipentene inhibited the growth of lung cancer cells and suppressed the growth of transplanted tumors in nude mice. Expression of apoptosis and autophagy-related genes were increased in tumors after treatment with (+)-Dipentene. Furthermore, the use of chloroquine, an autophagy inhibitor, and knockdown of the atg5 gene, suppressed the apoptosis induced by (+)-Dipentene[6].


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